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Results 21-30 of 34 (Search time: 0.002 seconds).
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Issue Date
Title
Author(s)
2005
In vitro sensitivity to imatinib-induced inhibition of ABL kinase activity is predictive of molecular response in patients with de-novo CML.
White, D.
;
Saunders, V.
;
Lyons, A.
;
Branford, S.
;
Grigg, A.
;
To, L.
;
Hughes, T.
2006
OCT-1-mediated influx is a key determinant of the intraceflular uptake of imatinib but not nilotinib, (AMN107): reduced OCT-1 activity is the cause of low in vitro sensitivity to imatinib
White, D.
;
Saunders, V.
;
Dang, P.
;
Engler, J.
;
Zannettino, A.
;
Cambareri, A.
;
Quinn, S.
;
Manley, P.
;
Hughes, T.
2009
Chronic myeloid leukemia: An update of concepts and management recommendations of European LeukemiaNet
Baccarani, M.
;
Cortes, J.
;
Pane, F.
;
Niederwieser, D.
;
Saglio, G.
;
Apperley, J.
;
Cervantes, F.
;
Deininger, M.
;
Gratwohl, A.
;
Guilhot, F.
;
Hochhaus, A.
;
Horowitz, M.
;
Hughes, T.
;
Kantarjian, H.
;
Larson, R.
;
Radich, J.
;
Simonsson, B.
;
Silver, R.
;
Goldman, J.
;
Hehlmann, R.
2008
Long-term imatinib therapy promotes bone formation in CML patients
Fitter, S.
;
Dewar, A.
;
Kostakis, P.
;
To, L.
;
Hughes, T.
;
Roberts, M.
;
Lynch, K.
;
Vernon-Roberts, B.
;
Zannettino, A.
2008
Dasatinib suppresses in vitro natural killer cell cytotoxicity
Blake, S.
;
Lyons, A.
;
Fraser, C.
;
Hayball, J.
;
Hughes, T.
2006
Imatinib as a potential antiresorptive therapy for bone disease
Dewar, A.
;
Farrugia, A.
;
Condina, M.
;
To, L.
;
Hughes, T.
;
Vernon-Roberts, B.
;
Zannettino, A.
2009
Dasatinib in the Treatment of Chronic Myeloid Leukemia in Accelerated Phase After Imatinib Failure: The START A Trial
Apperley, J.
;
Cortes, J.
;
Kim, D.
;
Roy, L.
;
Roboz, G.
;
Rosti, G.
;
Bullorsky, E.
;
Abruzzese, E.
;
Hochhaus, A.
;
Heim, D.
;
De Souza, C.
;
Larson, R.
;
Lipton, J.
;
Khoury, J.
;
Kim, H.
;
Sillaber, C.
;
Hughes, T.
;
Erben, P.
;
Tornout, J.
;
Stone, R.
2004
Real-time quantitative PCR analysis can be used as a primary screen to identify patients with CML treated with imatinib who have BCR-ABL kinase domain mutations
Branford, S.
;
Rudzki, Z.
;
Parkinson, I.
;
Grigg, A.
;
Taylor, K.
;
Seymour, J.
;
Durrant, S.
;
Browett, P.
;
Schwarer, A.
;
Arthur, C.
;
Catalano, J.
;
Leahy, M.
;
Filshie, R.
;
Bradstock, K.
;
Herrmann, R.
;
Joske, D.
;
Lynch, K.
;
Hughes, T.
2007
Most CML patients who have a suboptimal response to imatinib have low OCT-1 activity: higher doses of imatinib may overcome the negative impact of low OCT-1 activity
White, D.
;
Saunders, V.
;
Dang, P.
;
Engler, J.
;
Venables, A.
;
Zrim, S.
;
Zannettino, A.
;
Lynch, K.
;
Manley, P.
;
Hughes, T.
2007
Imatinib increases the intracellular concentration of nilotinib which may explain the observed synergy between these drugs
White, D.
;
Saunders, V.
;
Quinn, S.
;
Manley, P.
;
Hughes, T.
Discover
Author
11
Branford, S.
10
Hochhaus, A.
8
Zannettino, A.
7
Dewar, A.
7
Grigg, A.
7
To, L.
7
White, D.
6
Lynch, K.
6
Lyons, A.
6
Saunders, V.
.
next >
Subject
30
Benzamides
30
Imatinib Mesylate
30
Piperazines
26
Leukemia, Myelogenous, Chronic, B...
17
Antineoplastic Agents
17
Fusion Proteins, bcr-abl
12
Adult
12
Female
12
Male
12
Protein Kinase Inhibitors
.
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