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Results 31-40 of 184 (Search time: 0.002 seconds).
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PreviewIssue DateTitleAuthor(s)
1998Haemolytic-uraemic syndrome outbreak caused by Escherichia coli O111:H-: clinical outcomesHenning, P.; Tham, E.; Martin, A.; Beare, T.; Jureidini, K.
1999Assignment of the Uroplakin 1b (Upk1b) gene to mouse Chromosome 16 bands B5-C2 by in situ hybridzationWebb, G.; Finch, J.; Cowled, P.
1995Fatigue of jaw muscles and speech mechanismsMiles, T.; Nordstrom, M.; Gandevia, S.C.; Enoka, R.M.; McComas, A.J.; Stuart, D.G.; Thomas, C.K.
1996Physiological Levels of Calcitonin Regulate the Mouse Osteoclast Calcitonin Receptor by a Protein Kinase A-Mediated MechanismWada, S.; Udagawa, N.; Nagata, N.; Martin, T.; Findlay, D.
1999Identification of a 14-3-3 binding sequence in the common B chain of the granulocyte-macrophage colony-stimulating factor (GM-CSF), interleukin-3 (IL-3), and IL-5 receptors that is serine-phosphorylated by GM-CSFStomski, F.; Dottore, M.; Winnall, W.; Guthridge, M.; Woodcock, J.; Bagley, C.; Thomas, D.; Andrews, R.; Berndt, M.; Lopez, A.
1995Special department Correspondence [Gastrocystoplasty]Dewan, P.; Chacko, J.; Ashwood, P.
1997Enhancement of Neutrophil-Mediated Killing of Plasmodium Falciparum Asexual Blood Forms By Fatty Acids : Importance of Fatty Acid StructureKumaratilake, L.; Ferrante, A.; Robinson, B.; Jaeger, T.; Poulos, A.
1996Homologous Regulation of the Rat C1a Calcitonin Receptor (CTR) in Nonosteoclastic Cells is Independent of CTR Messenger Ribonucleic Acid Changes and Cyclis Adenosine 3', 5' -Monophospate-Dependent Protein Kinase ActivationFindlay, D.; Houssami, S.; Christopoulos, G.; Sexton, P.
1997Crucial role of the residue at the F'-G' loop of the human GM-CSF receptor a chain for ligand recognition.Rajotte, D.; Cadieux, C.; Haman, A.; Wilkes, B.; Clark, S.; Hercus, T.; Woodcock, J.; Lopez, A.; Hoang, T.
1998Identification of a human HECT family protein with homology to the Drosophila tumor suppressor gene hyperplastic discsCallaghan, M.; Russell, A.; Woollatt, E.; Sutherland, G.; Sutherland, R.; Watts, C.

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