Please use this identifier to cite or link to this item: https://hdl.handle.net/2440/107488
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dc.contributor.authorLee, K.-
dc.contributor.authorIreland, K.-
dc.contributor.authorBleeze, M.-
dc.contributor.authorShoubridge, C.-
dc.date.issued2017-
dc.identifier.citationNeuroscience, 2017; 357:220-231-
dc.identifier.issn0306-4522-
dc.identifier.issn1873-7544-
dc.identifier.urihttp://hdl.handle.net/2440/107488-
dc.description.abstractAbstract not available-
dc.description.statementofresponsibilityK. Lee, K. Ireland, M. Bleeze and C. Shoubridge-
dc.language.isoen-
dc.publisherElsevier BV-
dc.rights© 2017 IBRO. Published by Elsevier Ltd. All rights reserved.-
dc.source.urihttp://dx.doi.org/10.1016/j.neuroscience.2017.06.010-
dc.subjectARX; intellectual disability; epilepsy; interneuron migration; calbindin interneurons; polyalanine expansion mutations-
dc.titleARX polyalanine expansion mutations lead to migration impediment in the rostral cortex coupled with a developmental deficit of calbindin-positive cortical GABAergic interneurons-
dc.typeJournal article-
dc.identifier.doi10.1016/j.neuroscience.2017.06.010-
dc.relation.granthttp://purl.org/au-research/grants/nhmrc/1063025-
dc.relation.granthttp://purl.org/au-research/grants/arc/FT120100086-
pubs.publication-statusPublished-
dc.identifier.orcidShoubridge, C. [0000-0002-0157-3084]-
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Paediatrics publications

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