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PreviewIssue DateTitleAuthor(s)
2013Substance P as a mediator of neurogenic inflammation after balloon compression induced spinal cord injuryLeonard, A.; Thornton, E.; Vink, R.
2014The neuroprotective activity of the amyloid precursor protein against traumatic brain injury is mediated via the heparin binding site in residues 96-110Corrigan, F.; Thornton, E.; Roisman, L.; Leonard, A.; Vink, R.; Blumbergs, P.; Van Den Heuvel, C.; Cappai, R.
2016Neurogenic inflammation after traumatic brain injury and its potentiation of classical inflammationCorrigan, F.; Mander, K.; Leonard, A.; Vink, R.
2012A Substance P Antagonist Improves Outcome in Female Sprague Dawley Rats Following Diffuse Traumatic Brain InjuryCorrigan, F.; Leonard, A.; Ghabriel, M.; Van Den Heuvel, C.; Vink, R.
2010Kinin receptor antagonists as potential neuroprotective agents in central nervous system injuryThornton, E.; Ziebell, J.; Leonard, A.; Vink, R.
2013The effect of an NK1 receptor antagonist on blood spinal cord barrier permeability following balloon compression-induced spinal cord injuryLeonard, A.; Vink, R.; Katayama, Y.; Maeda, T.; Kuroiwa, T.
2013Substance P antagonists as a novel intervention for brain edema and raised intracranial pressureGabrielian, L.; Helps, S.; Thornton, E.; Turner, R.; Leonard, A.; Vink, R.; Katayama, Y.; Maeda, T.; Kuroiwa, T.
2014Changes in substance P and NK1 receptor immunohistochemistry following human spinal cord injuryLeonard, A.; Manavis, J.; Blumbergs, P.; Vink, R.