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PreviewIssue DateTitleAuthor(s)
2003Neuropeptide release influences brain edema formation after diffuse traumatic brain injuryVink, R.; Young, A.; Bennett, C.; Hu, X.; O'Connor, C.; Cernak, I.; Nimmo, A.; Kuroiwa, T.; Baethmann, A.; Czernicki, Z.; Hoff, J.T.; Ito, U.; Katayama, Y.; Mararou, A.; Mendelow, A.D.; Reulen, H.J.
2014Treatment with the NK1 antagonist emend reduces blood brain barrier dysfunction and edema formation in an experimental model of brain tumorsHarford-Wright, E.; Lewis, K.; Ghabriel, M.; Vink, R.; Alonso, M.
2006The temporal profile of edema formation differs between male and female rats following diffuse traumatic brain injuryO'Connor, C.; Cernak, I.; Vink, R.; Hoff, J.; Keep, R.; Xi, G.; Hua, Y.; International Symposium on Brain Edema and Tissue Injury (13th : 2005 : Ann Arbor, Mich.)
2012Combined tissue plasminogen activator and an NK1 tachykinin receptor antagonist: an effective treatment for reperfusion injury following acute ischemic stroke in ratsTurner, R.; Vink, R.
2012Targeting classical but not neurogenic inflammation reduces peritumoral oedema in secondary brain tumoursLewis, K.; Harford-Wright, E.; Vink, R.; Ghabriel, M.
2012Treatment with a substance P receptor antagonist is neuroprotective in the intrastriatal 6-hydroxydopamine model of early Parkinson's DiseaseThornton, E.; Vink, R.; Block, M.L.
2011Towards drug discovery for brain tumours: interaction of kinins and tumours at the blood brain barrier interfaceHarford-Wright, E.; Lewis, K.; Vink, R.

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