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PreviewIssue DateTitleAuthor(s)
2014Treatment with the NK1 antagonist emend reduces blood brain barrier dysfunction and edema formation in an experimental model of brain tumorsHarford-Wright, E.; Lewis, K.; Ghabriel, M.; Vink, R.; Alonso, M.
2013Walker 256 tumour cells increase substance P immunoreactivity locally and modify the properties of the blood-brain barrier during extravasation and brain invasionLewis, K.; Harford-Wright, E.; Vink, R.; Nimmo, A.; Ghabriel, M.
2011Characterizing the role of the neuropeptide substance P in experimental subarachnoid hemorrhageBarry, C.; Helps, S.; Van Den Heuvel, C.; Vink, R.
2012Targeting classical but not neurogenic inflammation reduces peritumoral oedema in secondary brain tumoursLewis, K.; Harford-Wright, E.; Vink, R.; Ghabriel, M.
2012Substance P-induced changes in cell genesis following diffuse traumatic brain injuryCarthew, H.; Ziebell, J.; Vink, R.
2010Angiotensin-converting enzyme (ACE) inhibitors exacerbate histological damage and motor deficits after experimental traumatic brain injuryHarford-Wright, E.; Thornton, E.; Vink, R.
2012A Substance P Antagonist Improves Outcome in Female Sprague Dawley Rats Following Diffuse Traumatic Brain InjuryCorrigan, F.; Leonard, A.; Ghabriel, M.; Van Den Heuvel, C.; Vink, R.
2012Treatment with a substance P receptor antagonist is neuroprotective in the intrastriatal 6-hydroxydopamine model of early Parkinson's DiseaseThornton, E.; Vink, R.; Block, M.L.
2013The effect of an NK1 receptor antagonist on blood spinal cord barrier permeability following balloon compression-induced spinal cord injuryLeonard, A.; Vink, R.; Katayama, Y.; Maeda, T.; Kuroiwa, T.

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