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PreviewIssue DateTitleAuthor(s)
2001Production of chemokines in vivo in response to microbial stimulationCoates, N.; McColl, S.
2006CXCL16 regulates cell-mediated immunity to Salmonella enterica serovar Enteritidis via promotion of gamma interferon productionFahy, O.; Townley, S.; McColl, S.
2003A role for macrophage inflammatory protein-3 a/CC chemokine ligand 20 in immune priming during T cell-mediated inflammation of the central nervous systemKohler, R.; Caon, A.; Willenborg, D.; Clark-Lewis, I.; McColl, S.
2004Inhibition of generation of cytotoxic T lymphocyte activity by a CCL19/macrophage inflammatory protein (MIP)-3beta antagonistPilkington, K.; Clark-Lewis, I.; McColl, S.
2004Expression of rat I-TAC/CXCL11/SCYA11 during central nervous system inflammation: comparison with other CXCR3 ligandsMcColl, S.; Mahalingam, S.; Staykova, M.; Tylaska, L.; Fisher, K.; Strick, C.; Gladue, R.; Neote, K.; Willenborg, D.
2008Antagonism of the chemokine receptors CXCR3 and CXCR4 reduces the pathology of experimental autoimmune encephalomyelitisKohler, R.; Comerford, I.; Townley, S.; Haylock-Jacobs, S.; Clark-Lewis, I.; McColl, S.
2008Toxocara canis larval excretory/secretory proteins impair eosinophil-dependent resistance of mice to Nippostrongylus brasiliensisGiacomin, P.; Cava, M.; Tumes, D.; Gauld, A.; Iddawela, D.; McColl, S.; Parsons, J.; Gordon, D.; Dent, L.
2009Inhibition of CCR6 Function Reduces the Severity of Experimental Autoimmune Encephalomyelitis via Effects on the Priming Phase of the Immune ResponseListon, A.; Kohler, R.; Townley, S.; Haylock-Jacobs, S.; Comerford, I.; Caon, A.; Webster, J.; Harrison, J.; Swann, J.; Clark-Lewis, I.; Korner, H.; McColl, S.
2007IFNγ differentially controls the development of idiopathic pneumonia syndrome and GVHD of the gastrointestinal tractBurman, A.; Banovic, T.; Kuns, R.; Clouston, A.; Stanley, A.; Morris, E.; Rowe, V.; Bofinger, H.; Skoczylas, R.; Raffelt, N.; Fahy, O.; McColl, S.; Engwerda, C.; Mcdonald, K.; Hill, G.
2007CC chemokine ligand-5 (CCL5/RANTES) and CC chemokine ligand-18 (CCL18/PARC) are specific markers of refractory unstable angina pectoris and are transiently raised during severe ischemic symptomsKraaijeveld, A.; de Jager, S.; de Jager, W.; Prakken, B.; McColl, S.; Haspels, I.; Putter, H.; van Berkel, T.; Nagerlkerken, L.; Jukema, J.; Biessen, E.