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PreviewIssue DateTitleAuthor(s)
2008Interaction with factor inhibiting HIF-1 defines an additional mode of cross-coupling between the Notch and hypoxia signaling pathwaysZheng, X.; Karttunen, S.; Dias, J.; Zheng, X.; Gradin, K.; Wallis, T.; Hamilton, B.; Gustafsson, M.; Ruas, J.; Wilkins, S.; Bilton, R.; Brismar, K.; Whitelaw, M.; Pereira, T.; Gorman, J.; Ericson, J.; Peet, D.; Lendahl, U.; Poellinger, L.
1999Degradation of the basic helix-loop-helix/Per-ARNT-Sim homology domain dioxin receptor via the ubiquitin/proteasome pathwayRoberts, B.; Whitelaw, M.
2007Differential activation of insulin receptor substrates 1 and 2 by insulin-like growth factor-activated insulin receptorsDenley, A.; Carroll, J.; Brierley, G.; Cosgrove, L.; Wallace, J.; Forbes, B.; Roberts, C.
2005Transactivation of CXCR4 by the insulin-like growth factor-1 receptor (IGF-1R) in human MDA-MB-231 breast cancer epithelial cellsAkekawatchai, C.; Holland, J.; Kochetkova, M.; Wallace, J.; McColl, S.
2003Defining the role for XAP2 in stabilization of the dioxin receptorLees, M.; Peet, D.; Whitelaw, M.
2001The protein tyrosine phosphatase TCPTP suppresses the tumorigenicity of glioblastoma cells expressing a mutant epidermal growth factor receptorKlingler-Hoffmann, M.; Fodero-Tavoletti, M.; Mishima, K.; Narita, Y.; Cavenee, W.; Furnari, F.; Huang, H.; Tiganis, T.
2005Activity of hypoxia-inducible factor 2α is regulated by association with the NF-κB essential modulatorBracken, C.; Whitelaw, M.; Peet, D.
2009Activation of the Akt-NF-kappa B Pathway by Subtilase Cytotoxin through the ATF6 Branch of the Unfolded Protein ResponseYamazaki, H.; Hiramatsu, K.; Hayakawa, K.; Tagawa, Y.; Okamura, M.; Ogata, R.; Huang, T.; Nakajima, S.; Yao, J.; Paton, A.; Paton, J.; Kitamura, M.
2012Unfolded protein response causes a phenotypic shift of inflamed glomerular cells toward redifferentiation through dual blockade of Akt and Smad signaling pathwaysJohno, H.; Nakajima, S.; Kato, H.; Yao, J.; Paton, A.; Paton, J.; Katoh, R.; Shimizu, F.; Kitamura, M.
2001Peptide insertions in domain 4 of hbc, the shared signalling receptor subunit for GM-CSF, IL3 and IL5, induce ligand-independent activationJones, K.; Bagley, C.; Butcher, C.; Barry, S.; Vadas, M.; D'Andrea, R.