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Results 1-10 of 12 (Search time: 0.002 seconds).
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PreviewIssue DateTitleAuthor(s)
1995Activating point mutations in the common beta subunit of the human GM-CSF, IL-3 and IL-5 receptors suggest the involvement of beta subunit dimerization and cell type-specific molecules in signallingJenkins, B.; D'Andrea, R.; Gonda, T.
1999Simultaneous antagonism of interleukin-5, granulocyte-macrophage colony-stimulting factor, and interleukin-3 stimulation of human eosinophils by targetting the common cytokine binding site of their receptorsSun, Q.; Jones, K.; McClure, B.; Cambareri, B.; Zacharakis, B.; Iversen, P.; Stomski, F.; Woodcock, J.; Bagley, C.; D'Andrea, R.; Lopez, A.
1997Activating mutations in cytokine receptors: implications for receptor function and role in disease.Gonda, T.; D'Andrea, R.
2000A model for assembly and activation of the GM-CSF, IL-3 and IL-5 receptors: insights from activated mutants of the common b subunitD'Andrea, R.; Gonda, T.
2001GM-CSF binding to its receptor induces oligomerisation of the common beta-subunitMc Clure, B.; Woodcock, J.; Harrison-Findik, D.; Lopez, A.; D'Andrea, R.
2002The nucleotide-binding site of human sphingosine kinase 1Pitson, S.; Moretti, P.; Zebol, J.; Zareie, R.; Derian, C.; Darrow, A.; Qi, J.; D'Andrea, R.; Bagley, C.; Vadas, M.; Wattenberg, B.
2000An oncogenic role of sphingosine kinaseXia, P.; Gamble, J.; Wang, L.; Pitson, S.; Moretti, P.; Wattenberg, B.; D'Andrea, R.; Vadas, M.
2004Constitutive mutants of the GM-CSF receptor reveal multiple pathways leading to myeloid cell survival, proliferation, and granulocyte-macrophage differentiationBrown, A.; Peters, M.; D'Andrea, R.; Gonda, T.
1998Dysregulated hematopoiesis and a progressive neurological disorder induced by expression of an activated form of the human common β chain in transgenic miceD'Andrea, R.; Harrison-Findik, D.; Butcher, C.; Finnie, J.; Blumbergs, P.; Bartley, P.; McCormack, M.; Jones, K.; Rowland, R.; Gonda, T.; Vadas, M.
2018Differential effects on gene transcription and hematopoietic differentiation correlate with GATA2 mutant disease phenotypesChong, C.-E.; Venugopal, P.; Stokes, P.; Lee, Y.; Brautigan, P.; Yeung, D.; Babic, M.; Engler, G.; Lane, S.; Klingler-Hoffmann, M.; Matthews, J.; D'Andrea, R.; Brown, A.; Hahn, C.; Scott, H.