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PreviewIssue DateTitleAuthor(s)
2009Targeted treatment of imatinib-resistant chronic myeloid leukemia: Focus on dasatinibChuah, C.; Vaz de Melo, J.
2009Imatinib sensitivity as a consequence of a CSF1R-Y571D mutation and CSF1/CSF1R signaling abnormalities in the cell line GDM1Chase, A.; Schultheis, B.; Kreil, S.; Baxter, J.; Hidalgo-Curtis, C.; Jones, A.; Zhang, L.; Grand, F.; Vaz de Melo, J.; Cross, N.
2009Proteasome proteolytic profile is linked to Bcr-Abl expressionCrawford, L.; Windrum, P.; Magill, L.; Vaz de Melo, J.; McCallum, L.; McMullin, M.; Ovaa, H.; Walker, B.; Irvine, A.
2009CD40 and B-cell receptor signalling induce MAPK family members that can either induce or repress Bcl-6 expressionBatlle, A.; Papadopoulou, V.; Gomes, A.; Willimott, S.; Vaz de Melo, J.; Naresh, K.; Lam, E.; Wagner, S.
2009The impact of gene profiling in chronic myeloid leukaemiaYong, A.; Vaz de Melo, J.
2009Metabolic characteristics of imatinib resistance in chronic myeloid leukaemia cellsKlawitter, J.; Kominsky, D.; Brown, J.; Klawitter, J.; Christians, U.; Leibfritz, D.; Vaz de Melo, J.; Eckhardt, S.; Serkova, N.
2009Selecting optimal second-line tyrosine kinase inhibitor therapy for chronic myeloid leukemia patients after imatinib failure: does the BCR-ABL mutation status really matter?Branford, S.; Vaz de Melo, J.; Hughes, T.
2009Abnormalities in glucose uptake and metabolism in imatinib-resistant human BCR-ABL positive cellsKominsky, D.; Klawitter, J.; Brown, J.; Boros, L.; Vaz de Melo, J.; Eckhardt, S.; Serkova, N.
2009The level of BCR-ABL1 kinase activity before treatment does not identify myeloid leukemia patients who fail to achieve a complete cytogenetic response on imatinibKhorashad, J.; Wagner, S.; Greener, L.; Marin, D.; Reid, A.; Milojkovic, D.; Patel, H.; Willimott, S.; Rezvani, K.; Gerrard, G.; Loaiza, S.; Davis, J.; Goldman, J.; Vaz de Melo, J.; Apperley, J.; Foroni, L.

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