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Results 1-10 of 10 (Search time: 0.002 seconds).
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Issue Date
Title
Author(s)
2013
Safety and efficacy of imatinib cessation for CML patients with stable undetectable minimal residual disease: results from the TWISTER study
Ross, D.
;
Branford, S.
;
Seymour, J.
;
Schwarer, A.
;
Arthur, C.
;
Yeung, D.
;
Dang, P.
;
Goyne, J.
;
Slader, C.
;
Filshie, R.
;
Mills, A.
;
Vaz de Melo, J.
;
White, D.
;
Grigg, A.
;
Hughes, T.
2013
Association between imatinib transporters and metabolizing enzymes genotype and response in newly diagnosed chronic myeloid leukemia patients receiving imatinib therapy
Angelini, S.
;
Soverini, S.
;
Ravegnini, G.
;
Barnett, M.
;
Turrini, E.
;
Thornquist, M.
;
Pane, F.
;
Hughes, T.
;
White, D.
;
Radich, J.
;
Kim, D.
;
Saglio, G.
;
Cilloni, D.
;
Iacobucci, I.
;
Perini, G.
;
Woodman, R.
;
Cantelli-Forti, G.
;
Baccarani, M.
;
Hrelia, P.
;
Martinelli, G.
2013
Proton pump inhibitors significantly increase the intracellular concentration of nilotinib, but not imatinib in target CML cells
White, D.
;
Eadie, L.
;
Saunders, V.
;
Hiwase, D.
;
Hughes, T.
2003
Successful peripheral blood stem cell mobilisation with filgrastim in patients with chronic myeloid leukaemia achieving complete cytogenetic response with imatinib, without increasing disease burden as measured by quantitative real-time PCR
Hui, C.
;
Goh, K.
;
White, D.
;
Branford, S.
;
Grigg, A.
;
Seymour, J.
;
Kwan, Y.
;
Walsh, S.
;
Hoyt, R.
;
Trickett, A.
;
Rudzki, Z.
;
Ma, D.
;
To, L.
;
Hughes, T.
2010
The poor response to imatinib observed in CML patients with low OCT-1 activity is not attributable to lower uptake of imatinib into their CD34⁺ cells
Engler, J.
;
Frede, A.
;
Saunders, V.
;
Zannettino, A.
;
White, D.
;
Hughes, T.
2006
OCT-1-mediated influx is a key determinant of the intraceflular uptake of imatinib but not nilotinib, (AMN107): reduced OCT-1 activity is the cause of low in vitro sensitivity to imatinib
White, D.
;
Saunders, V.
;
Dang, P.
;
Engler, J.
;
Zannettino, A.
;
Cambareri, A.
;
Quinn, S.
;
Manley, P.
;
Hughes, T.
2010
Plasma adiponectin levels are markedly elevated in imatinib-treated chronic myeloid leukemia (CML) patients: A mechanism for improved insulin sensitivity in Type 2 diabetic CML patients?
Fitter, S.
;
Vandyke, K.
;
Schultz, C.
;
White, D.
;
Hughes, T.
;
Zannettino, A.
2007
Most CML patients who have a suboptimal response to imatinib have low OCT-1 activity: higher doses of imatinib may overcome the negative impact of low OCT-1 activity
White, D.
;
Saunders, V.
;
Dang, P.
;
Engler, J.
;
Venables, A.
;
Zrim, S.
;
Zannettino, A.
;
Lynch, K.
;
Manley, P.
;
Hughes, T.
2011
SHP-1 expression accounts for resistance to imatinib treatment in Philadelphia chromosome-positive cells derived from patients with chronic myeloid leukemia
Esposito, N.
;
Colavita, I.
;
Quintarelli, C.
;
Sica, A.
;
Peluso, A.
;
Luciano, L.
;
Picardi, M.
;
Vecchio, L.
;
Buonomo, T.
;
Hughes, T.
;
White, D.
;
Radich, J.
;
Russo, D.
;
Branford, S.
;
Saglio, G.
;
Vaz de Melo, J.
;
Martinelli, R.
;
Ruoppolo, M.
;
Kalebic, T.
;
Martinelli, G.
;
et al.
2008
Dasatinib cellular uptake and efflux in chronic myeloid leukemia cells: therapeutic implications
Hiwase, D.
;
Saunders, V.
;
Hewett, D.
;
Frede, A.
;
Zrim, S.
;
Dang, P.
;
Eadie, L.
;
To, L.
;
Vaz de Melo, J.
;
Kumar, S.
;
Hughes, T.
;
White, D.
Discover
Author
5
Saunders, V.
4
Dang, P.
4
Zannettino, A.
3
Branford, S.
3
Engler, J.
3
Vaz de Melo, J.
2
Eadie, L.
2
Frede, A.
2
Grigg, A.
2
Hiwase, D.
.
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Subject
10
Piperazines
10
Pyrimidines
7
Antineoplastic Agents
6
Female
6
Male
5
Adult
5
Protein Kinase Inhibitors
5
Treatment Outcome
4
Middle Aged
3
Aged
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