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Results 1-10 of 162 (Search time: 0.003 seconds).
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PreviewIssue DateTitleAuthor(s)
2001Hormone status selects for spontaneous somatic androgen receptor variants that demonstrate specific ligand and cofactor dependent activities in autochthonous prostate cancerHan, G.; Foster, B.; Mistry, S.; Buchanan, G.; Harris, J.; Tilley, W.; Greenberg, N.
2001Mutations at the boundary of the hinge and ligand binding domain of the androgen receptor confer increased transactivation functionBuchanan, G.; Yang, M.; Harris, J.; Nahm, H.; Han, G.; Moore, N.; Bentel, J.; Matusik, R.; Horsfall, D.; Marshall, V.; Greenberg, N.; Tilley, W.
2006Suppression of androgen receptor signaling in prostate cancer cells by an inhibitory receptor variantButler, L.; Centenera, M.; Neufing, P.; Buchanan, G.; Choong, C.; Ricciardelli, C.; Saint, K.; Lee, M.; Ochnik, A.; Yang, M.; Brown, M.; Tilley, W.
2002Juxtamembrane mutant V560GKit is more sensitive to Imatinib (STI571) compared with wild-type c-kit whereas the kinase domain mutant D816VKit is resistantFrost, M.; Ferrao, P.; Hughes, T.; Ashman, L.
2004The apical caspase dronc governs programmed and unprogrammed cell death in DrosophilaChew, S.; Akdemir, F.; Chen, P.; Lu, W.; Mills, K.; Daish, T.; Kumar, S.; Rodriguez, A.; Abrams, J.
2004Drosophila caspase DRONC is required for specific developmental cell death pathways and stress-induced apoptosisDaish, T.; Mills, K.; Kumar, S.
2002Sphingosine kinase interacts with TRAF2 and dissects tumor necrosis factor-a signalingXia, P.; Wang, L.; Moretti, P.; Albanese, N.; Chai, F.; Pitson, S.; D'Andrea, R.; Gamble, J.; Vadas, M.
1997Expression of activated mutants of the human interleukin-3/interleukin-5/granulocyte-macrophage colony-stimulating factor receptor common beta subunit in primary hematopoietic cells induces factor-independent proliferation and differentiation.McCormack, M.; Gonda, T.
2001Collocation of androgen receptor gene mutations in prostate cancerBuchanan, G.; Greenberg, N.; Scher, H.; Harris, J.; Marshall, V.; Tilley, W.
2016EPHA2 mutations contribute to congenital cataract through diverse mechanismsDave, A.; Martin, S.; Kumar, R.; Craig, J.; Burdon, K.; Sharma, S.