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Results 11-20 of 48 (Search time: 0.004 seconds).
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PreviewIssue DateTitleAuthor(s)
2015Structure-Activity Relationships of (+)-Naltrexone-Inspired Toll-like Receptor 4 (TLR4) AntagonistsSelfridge, B.R.; Wang, X.; Zhang, Y.; Yin, H.; Grace, P.M.; Watkins, L.R.; Jacobson, A.E.; Rice, K.C.
2013Rifampin inhibits Toll-like receptor 4 signaling by targeting myeloid differentiation protein 2 and attenuates neuropathic painWang, X.; Grace, P.; Pham, M.; Cheng, K.; Strand, K.; Smith, C.; Li, J.; Watkins, L.; Yin, H.
1999Calcitonin Receptor-Mediated Growth Suppression of HEK-293 Cells is Accompanied by Induction of p21 WAF1/CIP1 and G2/M ArrestEvdokiou, A.; Raggatt, L.; Findlay, D.; Atkins, G.
2021miR-542-3p Attenuates Bone Loss and Marrow Adiposity Following Methotrexate Treatment by Targeting sFRP-1 and Smurf2Zhang, Y.-L.; Liu, L.; Su, Y.-W.; Xian, C.J.
1998Casein kinase 2 phosphorylation of recombinant rat osteopontin enhances adhesion of osteoclasts but not osteoblastsKatayama, Y.; House, C.; Udagawa, N.; Kazama, J.; McFarland, R.; Martin, T.; Findlay, D.
2011An MD2 hot-spot-mimicking peptide that suppresses TLR4-mediated inflammatory response in vitro and in vivoLiu, L.; Ghosh, N.; Slivka, P.; Fiorini, Z.; Hutchinson, M.; Watkins, L.; Yin, H.
2009Activation of the Akt-NF-kappa B Pathway by Subtilase Cytotoxin through the ATF6 Branch of the Unfolded Protein ResponseYamazaki, H.; Hiramatsu, K.; Hayakawa, K.; Tagawa, Y.; Okamura, M.; Ogata, R.; Huang, T.; Nakajima, S.; Yao, J.; Paton, A.; Paton, J.; Kitamura, M.
2013Ocular expression and distribution of products of the POAG-associated chromosome 9p21 gene regionChidlow, G.; Wood, J.; Sharma, S.; Dimasi, D.; Burdon, K.; Casson, R.; Craig, J.; Mittal, B.
2012Unfolded protein response causes a phenotypic shift of inflamed glomerular cells toward redifferentiation through dual blockade of Akt and Smad signaling pathwaysJohno, H.; Nakajima, S.; Kato, H.; Yao, J.; Paton, A.; Paton, J.; Katoh, R.; Shimizu, F.; Kitamura, M.
2010αB-Crystallin inhibits the cell toxicity associated with amyloid fibril formation by κ-casein and the amyloid-β peptideDehle, F.; Ecroyd, H.; Musgrave, I.; Carver, J.

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