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PreviewIssue DateTitleAuthor(s)
2011Local delivery of GM-CSF protects mice from lethal pneumococcal pneumoniaSteinwede, K.; Tempelhof, O.; Bolte, K.; Maus, R.; Bohling, J.; Ueberberg, B.; Langer, F.; Christman, J.; Paton, J.; Ask, K.; Maharaj, S.; Kolb, M.; Gauldie, J.; Welte, T.; Maus, U.
2011The conformation and function of a multimodular glycogen-degrading pneumococcal virulence factorvan Bueren, A.; Ficko-Blean, E.; Pluvinage, B.; Hehemann, J.; Higgins, M.; Deng, L.; Ogunniyi, A.; Stroeher, U.; El Warry, N.; Burke, R.; Czjzek, M.; Paton, J.; Vocadlo, D.; Boraston, A.
2011In vivo leukocyte changes induced by Escherichia coli subtilase cytotoxinWang, H.; Paton, A.; McColl, S.; Paton, J.; Bliska, J.B.
2011Cathepsin G and neutrophil elastase play critical and nonredundant roles in lung-protective Immunity against Streptococcus pneumoniae in miceHahn, I.; Klaus, A.; Janze, A.; Steinwede, K.; Ding, N.; Bohling, J.; Brumshagen, C.; Serrano, H.; Gauthier, F.; Paton, J.; Welte, T.; Maus, U.; Weiser, J.N.
2011Pneumolysin with low hemolytic activity confers an early growth advantage to Streptococcus pneumoniae in the bloodHarvey, R.; Ogunniyi, A.; Chen, A.; Paton, J.; Camilli, A.
2011Contribution of serotype and genetic background to virulence of serotype 3 and serogroup 11 pneumococcal isolatesMcAllister, L.; Ogunniyi, A.; Stroeher, U.; Leach, A.; Paton, J.; Camilli, A.
2011Selective abrogation of BiP/GRP78 blunts activation of NF-κΒ through the ATF6 branch of the UPR: involvement of C/EBPβ and mTOR-dependent dephosphorylation of AktNakajima, S.; Hiramatsu, N.; Hayakawa, K.; Saito, Y.; Kato, H.; Huang, T.; Yao, J.; Paton, A.; Paton, J.; Kitamura, M.
2011A variable region within the genome of Streptococcus pneumoniae contributes to strain-strain variation in virulenceHarvey, R.; Stroeher, U.; Ogunniyi, A.; Smith-Vaughan, H.; Leach, A.; Paton, J.; Neyrolles, O.
2011A molecular mechanism for bacterial susceptibility to zincMcDevitt, C.; Ogunniyi, A.; Valkov, E.; Lawrence, M.; Kobe, B.; McEwan, A.; Paton, J.; Imlay, J.
2011Extracellular matrix formation enhances the ability of streptococcus pneumoniae to cause invasive diseaseTrappetti, C.; Ogunniyi, A.; Oggioni, M.; Paton, J.; Miyaji, E.N.

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