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https://hdl.handle.net/2440/8335
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DC Field | Value | Language |
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dc.contributor.author | Dekker, G. | - |
dc.contributor.author | Robillard, P. | - |
dc.date.issued | 2003 | - |
dc.identifier.citation | Journal of Reproductive Immunology, 2003; 59(2):245-251 | - |
dc.identifier.issn | 0165-0378 | - |
dc.identifier.issn | 1872-7603 | - |
dc.identifier.uri | http://hdl.handle.net/2440/8335 | - |
dc.description | Copyright © 2003 Elsevier Science Ireland Ltd. All rights reserved. | - |
dc.description.abstract | Recent Norwegian data suggest the presence of a causal relationship between prolonged birth intervals and the risk for preeclampsia in subsequent pregnancies. It has been proposed that the birth interval data explain the known association between a change in paternity and the risk for preeclampsia. In this review, the authors explore alternative explanations for the Norwegian findings, and as such argue that there is currently no reason to reject the primipaternity hypothesis. | - |
dc.description.statementofresponsibility | Gus Dekker, and Pierre-Yves Robillard | - |
dc.language.iso | en | - |
dc.publisher | Elsevier Sci Ireland Ltd | - |
dc.source.uri | http://www.sciencedirect.com/science/journal/01650378 | - |
dc.subject | Birth interval hypothesis | - |
dc.subject | Primipaternity hypothesis | - |
dc.subject | Preeclampsia | - |
dc.title | The birth interval hypothesis - does it really indicate the end of the primipaternity hypothesis | - |
dc.type | Journal article | - |
dc.identifier.doi | 10.1016/S0165-0378(03)00051-2 | - |
pubs.publication-status | Published | - |
dc.identifier.orcid | Dekker, G. [0000-0002-7362-6683] | - |
Appears in Collections: | Aurora harvest Obstetrics and Gynaecology publications |
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