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Please use this identifier to cite or link to this item: https://hdl.handle.net/2440/65488
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dc.contributor.authorEadie, L.-
dc.contributor.authorHughes, T.-
dc.contributor.authorWhite, D.-
dc.date.issued2010-
dc.identifier.citationLeukemia, 2010; 24(4):855-857-
dc.identifier.issn0887-6924-
dc.identifier.issn1476-5551-
dc.identifier.urihttp://hdl.handle.net/2440/65488-
dc.description.statementofresponsibilityL. Eadie, T. P. Hughes and D. L. White-
dc.language.isoen-
dc.publisherNature Publishing Group-
dc.rights© 2011 Nature Publishing Group, a division of Macmillan Publishers Limited. All Rights Reserved.-
dc.source.urihttp://dx.doi.org/10.1038/leu.2010.7-
dc.subjectTumor Cells, Cultured-
dc.subjectHumans-
dc.subjectBenzamides-
dc.subjectTetraethylammonium-
dc.subjectPiperazines-
dc.subjectPyrimidines-
dc.subjectPrazosin-
dc.subjectAdrenergic alpha-Antagonists-
dc.subjectProtein Kinase Inhibitors-
dc.subjectDrug Therapy, Combination-
dc.subjectOctamer Transcription Factor-1-
dc.subjectProtein-Tyrosine Kinases-
dc.subjectLeukemia, Myelogenous, Chronic, BCR-ABL Positive-
dc.subjectImatinib Mesylate-
dc.titleNilotinib does not significantly reduce imatinib OCT-1 activity in either cell lines or primary CML cells-
dc.typeJournal article-
dc.identifier.doi10.1038/leu.2010.7-
pubs.publication-statusPublished-
dc.identifier.orcidEadie, L. [0000-0003-1912-7602]-
dc.identifier.orcidHughes, T. [0000-0002-0910-3730] [0000-0002-7990-4509]-
dc.identifier.orcidWhite, D. [0000-0003-4844-333X]-
Appears in Collections:Agriculture, Food and Wine publications
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