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Please use this identifier to cite or link to this item: https://hdl.handle.net/2440/73782
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dc.contributor.authorRayner, C.-
dc.contributor.authorJones, K.-
dc.contributor.authorHorowitz, M.-
dc.contributor.editorParkman, H.-
dc.contributor.editorMcCallum, R.-
dc.date.issued2012-
dc.identifier.citationGastroparesis: Pathophysiology, Presentation and Treatment, 2012 / Parkman, H., McCallum, R. (ed./s), pp.177-190-
dc.identifier.isbn1607615517-
dc.identifier.isbn9781607615514-
dc.identifier.urihttp://hdl.handle.net/2440/73782-
dc.descriptionAlso has ISBN 1607615525 ; 9781607615521-
dc.description.abstractGastric emptying is frequently disordered in patients with long-standing type 1 and type 2 diabetes mellitus. While patients with diabetes present with similar symptoms to those with idiopathic gastroparesis, the relationship between gastric emptying and glycemic control represents an important additional consideration in the diabetic group. Specifically, acute variations in the blood glucose concentration affect the rate of gastric emptying, while conversely, gastric emptying is a key determinant of postprandial blood glucose. This has implications both for how gastroparesis should be defined in patients with diabetes, and for its optimal management.-
dc.description.statementofresponsibilityChristopher K. Rayner, Karen L. Jones and Michael Horowitz-
dc.language.isoen-
dc.publisherSpringer-
dc.source.urihttp://dx.doi.org/10.1007/978-1-60761-552-1_16-
dc.subjectDiabetes mellitus-
dc.subjectInterstitial cells of Cajal-
dc.subjectNitric oxide-
dc.subjectHeme oxygenase 1-
dc.subjectIncretin Glucagon-like peptide 1-
dc.subjectinsulin-
dc.subjectblood glucose concentration-
dc.titleDiabetic Gastroparesis-
dc.typeBook chapter-
dc.identifier.doi10.1007/978-1-60761-552-1_16-
dc.publisher.placeNew York-
pubs.publication-statusPublished-
dc.identifier.orcidRayner, C. [0000-0002-5527-256X]-
dc.identifier.orcidJones, K. [0000-0002-1155-5816]-
dc.identifier.orcidHorowitz, M. [0000-0002-0942-0306]-
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